Research

Our aim is to provide important contributions to the discovery and development of new drugs for a more efficient treatment of severe human diseases.  

Our group is developing novel selective compounds against major human diseases ( see overview), which are currently not or insufficiently treatable with available drugs. Each research project in guided by at least one of the following principles:
  • new chemical structures of the compounds
  • previously unexplored mechanisms of action
  • choice of the appropriate new target

Our expertise in drug development reaches from discovery of first “hits” to the in-vivo testing in the preclinical stage. Not only we are using state-of-the art methodology of medicinal and computational chemistry, but also we are developing appropriate and reliable biochemical and cellular assays in house to measure compound activity (e.g. P2-Subproject) . In addition, a novel MS/MS-based screening method is under development.

Whenever appropriate, we apply a rational approach (e.g. computer modelling in combination with virtual screening), followed by chemical synthesis of structurally interesting and promising new compounds and subsequent activity testing in vitro. Several cellular and non-cellular screening systems which have been developed by our group can be applied. We are using partially isolated protein extracts, cell free enzyme and membrane preparations as well as various mammalian cell lines, yeast and bacterial strains which, in many cases, have been transformed through recombinant expression of the respective human enzymes of interest.

All throughout the compound optimisation process, compounds are screened in parallel against highly homologous non-target enzymes to monitor selectivity.

Depending on the requirement of the project, compounds are optimised in iterative cycles comprising parallel synthesis, activity screening, molecular docking and modelling, and, not at the least, experience and instinct of our medicinal chemists.

Compounds which have been developed further and have proven to be highly active in vitro are subjected to in vivo tests. Further preclinical and clinical development is performed through cooperation with partners from industry. As an example, our original design of non-steroidal inhibitors of aromatase has successfully contributed to the development of selective compounds which are now in clinical use as first-line therapeutics against estrogen-dependent breast cancer in the U.S. after surgical treatment.

Project

Indication(s)

Scientific particularity

Compound mechanism of action

Discovery/ development strategy

P1:
Development of CYP 17 (P450 17) inhibitors for an improved anti-androgen treatment of prostate cancer

more informations

Prostate Cancer

Proven treatment strategy of blocking androgen action is exploited in a more efficient, novel way: inhibition of androgen production rather than receptor antagonists

Heme-complexing, steroid mimicking

No X-ray structure available; molecular modelling approach, combined with screening of focused in-house compound library

P2:
Development of CYP11B2 (aldosterone synthase) inhibitors for the treatment of heart failure, myocardial fibrosis, and hyperaldosteronism

more informations

P2-Subproject: assay development


Heart failure, myocardial fibrosis, and hyperaldosteronism

A new target was chosen by us to develop novel compounds with reduced side effects while relying on a proven therapeutic principle

Heme-complexing, steroid mimicking

No X-ray structure available; molecular modelling approach, combined with screening of focused in-house compound library
P3:
Novel Concept for Dual Inhibition of 5 α -Reductase

more informations

Benign Prostatic Hyperplasia (BPH)

Inhibition of both 5 α -Reductase isoforms by using a pro-drug concept (hybrid inhibitor); non-steroidal inhibitor structure will avoid side-effects attributable to the presently marketed steroid-like drugs.

Non-steroidal precursor drug (inhibitor of 5 α -Reductase type 1) is converted to the corresponding 5 α -Reductase type 2-inhibitor by prostatic esterases.

 


No X-ray structure available; molecular modelling approach, combined with screening of focused in-house compound library; pharmacokinetic evaluation
P4:
Selective inhibition of 17β-hydroxysteroid dehydrogenase type 1 (17β-HSD1): A promising approach for the treatment of hormone-dependent female disorders

more informations

Estrogen-dependent diseases such as breast cancer and endometriosis

Novel intracrinological approach aiming at high tissue specificity and low side-effects

Steroid mimicking competitive inhibitors devoid of estrogenicity

Rational design based on X-ray structure; differential docking of candidate compounds to estrogen receptor structure
P5:
Ligands of the human CD81 protein: Pharmacological tools that offer novel treatment options for hepatitis C virus infection

more informations

Hepatitis C virus infections

Novel target for Drug development; big medical need for specific antiviral treatment

Compounds compete with binding of viral E2 glycoprotein and prevent viral entry

Molecular modelling based on X-ray structure, virtual screening and rational design
P6:
Development of high-throughput screening (HTS) methods for compound libraries based on MS/MS detection

more informations

- not applicable -

Flexible, HTS compatible method for identifying ligands out of a complex mixture of compounds; main advantages: speed, label free

Compounds are identified based on affinity interaction with the target

- not applicable -
P7:
Development of novel non-ATP competitive drugs that inhibit protein kinases of the AGC family

more informations

Several forms of cancer, diabetes type-2

First small molecules rationally designed as allosteric effectors of the activity of a whole family of protein kinases

Binding to allosteric docking site modulates activity by conformational changes; non-ATP competitive properties anticipate better selectivity

Combined approach of peptide-guided strategy, library screening and virtual screening